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El GHK-Cu ejerce también efectos antiinflamatorios al inhibir la liberación de citoquinas proinflamatorias (IL-6, TNF-alfa) y al modular la actividad del factor nuclear NF-kB

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The present study, for the first time, demonstrates an underlying mechanism for the activation of eNOS by BPC 157 on stimulating the Cav-1 phosphorylation and reducing the binding of eNOS to Cav-1